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This is SCIENCE IN THE NEWS, in VOA Special English.The method combines an examination by positron emission tomography with a drug
that lights up beta-amyloid.The most widely held belief about the cause of Alzheimer's is that a protein, beta-
amyloid, builds up in patients' brains.The researchers say beta-amyloid destroys
communication links in the brain.Some scientists question whether beta-amyloid causes Alzheimer's disease.They
think that the protein build-up may result from it. But most researchers say thick
tangles or plaques of the protein are responsible for the condition.Plaques are
unusual clusters, or groups, of proteins.Scientists have been attempting for years to learn who may get Alzheimer's disease.Among older people, Alzheimer's is the most common form of dementia, the loss of
abilities needed for normal life.Doctors say methods to test the living have presented problems,
like high costs for widespread use.There is no cure for Alzheimer's, which steals
people's ability to care for themselves.Large amounts of this protein may destroy a
person's ability to think.A correct diagnosis, or identification, is important.The best way to diagnose the disease has been a medical examination of the brain
after a person dies.Scientists have been
working to produce a dependable test for the disease in the living.The United States' Food and Drug Administration is considering one such method.The PET device makes scans or images that doctors can
read.I'm Faith Lapidus.And I'm Bob Doughty.Today we tell about Alzheimer's disease.More than a
century after its discovery, Alzheimer's disease is still destroying people's brains.If the condition could be identified before its worst signs appear, people might get
at least temporary medical help.Other mental conditions may seem like Alzheimer's.Those conditions need medical treatment that is different from treatment for
Alzheimer's.But research may offer hope for the future.But treatment can slow its progress.Public and medical demand for a better way has been strong.
This is SCIENCE IN THE NEWS, in VOA Special English. I’m Faith Lapidus.
And I’m Bob Doughty. Today we tell about Alzheimer’s disease. More than a
century after its discovery, Alzheimer’s disease is still destroying people’s brains.
But research may offer hope for the future.
Scientists have been attempting for years to learn who may get Alzheimer’s disease.
If the condition could be identified before its worst signs appear, people might get
at least temporary medical help. There is no cure for Alzheimer’s, which steals
people’s ability to care for themselves. But treatment can slow its progress.
The most widely held belief about the cause of Alzheimer’s is that a protein, beta-
amyloid, builds up in patients’ brains. Large amounts of this protein may destroy a
person’s ability to think.
Some scientists question whether beta-amyloid causes Alzheimer’s disease. They
think that the protein build-up may result from it. But most researchers say thick
tangles or plaques of the protein are responsible for the condition. Plaques are
unusual clusters, or groups, of proteins. The researchers say beta-amyloid destroys
communication links in the brain.
Among older people, Alzheimer’s is the most common form of dementia, the loss of
abilities needed for normal life. Other mental conditions may seem like Alzheimer’s.
Those conditions need medical treatment that is different from treatment for
Alzheimer’s. A correct diagnosis, or identification, is important.
The best way to diagnose the disease has been a medical examination of the brain
after a person dies. Doctors say methods to test the living have presented problems,
like high costs for widespread use.
Public and medical demand for a better way has been strong. Scientists have been
working to produce a dependable test for the disease in the living.
The United States’ Food and Drug Administration is considering one such method.
The method combines an examination by positron emission tomography with a drug
that lights up beta-amyloid. The PET device makes scans or images that doctors can
read.
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